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mirna microarray  (CapitalBio Corporation)

 
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    Structured Review

    CapitalBio Corporation mirna microarray
    Table 1
    Mirna Microarray, supplied by CapitalBio Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/pcr-based+mirna+microarray+platform/mirna+microarray/pmc02682186-61-48-54
    Average 90 stars, based on 1 article reviews
    mirna microarray - by Bioz Stars, 2026-11
    90/100 stars

    Images

    1) Product Images from "Microarray profiling of microRNAs expressed in testis tissues of developing primates"

    Article Title: Microarray profiling of microRNAs expressed in testis tissues of developing primates

    Journal:

    doi: 10.1007/s10815-009-9305-y

    Table 1
    Figure Legend Snippet: Table 1

    Techniques Used:

    Related Articles

    other:

    Article Title: Regulatory effects of miR-28 on osteogenic differentiation of human bone marrow mesenchymal stem cells
    Article Snippet: TRIzol solution was from Invitrogen (USA), miRNA Isolation Kits were from Roche (Switzerland), and miRNA microarray V2.0 was purchased from Beijing CapitalBio Corporation (China; 576 miRNAs labeled in human, 238 in rats, and 358 in mice).

    Expressing:

    Article Title: Gene Expression Profiles of Circular RNAs and MicroRNAs in Chronic Rhinosinusitis With Nasal Polyps
    Article Snippet: .. MiRNA expression profile microarrays of these specimens were performed by CapitalBio. ..

    Control:

    Article Title: Oncogenic miR-20b-5p contributes to malignant behaviors of breast cancer stem cells by bidirectionally regulating CCND1 and E2F1.
    Article Snippet: CD44+CD24−/low BCSCs were isolated from MCF-7 and T47D cells by staining with CD44-APC, CD24-PE and ESA-FITC (BD Pharmingen, USA) antibodies via FACS as described in our previous research [8, 20]. miRNA profile and miRNA network Both human miRNA microarray fabrication and hybridization were performed as described previously [8]. .. The miRNA profiles of both BCSCs and control MCF-7 breast cancer cells were obtained from CapitalBio Corporation (Beijing, China). ..

    Purification:

    Article Title: MiR-672-5p-Mediated Upregulation of REEP6 in Spinal Dorsal Horn Participates in Bortezomib-Induced Neuropathic Pain in Rats.
    Article Snippet: Evidence shows that miRNAs are deeply involved in nervous system diseases, but whether miRNAs contribute to the bortezomib (BTZ)-induced neuropathic pain remains unclear.. We aimed to investigate whether miRNAs contribute to bortezomib (BTZ)-induced neuropathic pain and explore the related downstream cascades.. The level of miRNAs in the spinal dorsal horn was explored using miRNA microarray and PCR.

    Labeling:

    Article Title: MiR-672-5p-Mediated Upregulation of REEP6 in Spinal Dorsal Horn Participates in Bortezomib-Induced Neuropathic Pain in Rats.
    Article Snippet: Evidence shows that miRNAs are deeply involved in nervous system diseases, but whether miRNAs contribute to the bortezomib (BTZ)-induced neuropathic pain remains unclear.. We aimed to investigate whether miRNAs contribute to bortezomib (BTZ)-induced neuropathic pain and explore the related downstream cascades.. The level of miRNAs in the spinal dorsal horn was explored using miRNA microarray and PCR.

    Hybridization:

    Article Title: MiR-672-5p-Mediated Upregulation of REEP6 in Spinal Dorsal Horn Participates in Bortezomib-Induced Neuropathic Pain in Rats.
    Article Snippet: Evidence shows that miRNAs are deeply involved in nervous system diseases, but whether miRNAs contribute to the bortezomib (BTZ)-induced neuropathic pain remains unclear.. We aimed to investigate whether miRNAs contribute to bortezomib (BTZ)-induced neuropathic pain and explore the related downstream cascades.. The level of miRNAs in the spinal dorsal horn was explored using miRNA microarray and PCR.

    Microarray:

    Article Title: MiR-672-5p-Mediated Upregulation of REEP6 in Spinal Dorsal Horn Participates in Bortezomib-Induced Neuropathic Pain in Rats.
    Article Snippet: Evidence shows that miRNAs are deeply involved in nervous system diseases, but whether miRNAs contribute to the bortezomib (BTZ)-induced neuropathic pain remains unclear.. We aimed to investigate whether miRNAs contribute to bortezomib (BTZ)-induced neuropathic pain and explore the related downstream cascades.. The level of miRNAs in the spinal dorsal horn was explored using miRNA microarray and PCR.

    Article Title: MiR-26a-5p regulates proliferation, apoptosis, migration and invasion via inhibiting hydroxysteroid dehydrogenase like-2 in cervical cancer cell.
    Article Snippet: The total RNAs were isolated from 6 CC tissues and 6 PC tissues in according to the protocol of TRIzol reagent (15,596,026, Invitrogen, USA) and purified using mirVanaTM miRNA Isolation Kit (AM1561, Life Technologies, USA). .. Then, the differential expression of RNA was identified using miRNA microarray by CapitalBio Corporation (CapitalBio, China). ..

    Article Title: Retracted: Transcription activation of microRNA-25 by PEA3 augments progression of gastric cancer through suppressing SIK1.
    Article Snippet: .. A miRNA microarray analysis was performed by CapitalBio (Capital BioCorp, Beijing China) using tumor and the paired normal tissue samples from three patients. .. Total RNA from tissues was collected by TRIzol Reagent (Thermo Fisher Scientific Inc., Waltham, MA, USA).

    Article Title: miR-29b-3p Increases Radiosensitivity in Stemness Cancer Cells via Modulating Oncogenes Axis
    Article Snippet: .. Then, the microarray analysis of miRNAs was performed by CapitalBio Corporation following the protocol as previously described ( ). ..

    Quantitative Proteomics:

    Article Title: MiR-26a-5p regulates proliferation, apoptosis, migration and invasion via inhibiting hydroxysteroid dehydrogenase like-2 in cervical cancer cell.
    Article Snippet: The total RNAs were isolated from 6 CC tissues and 6 PC tissues in according to the protocol of TRIzol reagent (15,596,026, Invitrogen, USA) and purified using mirVanaTM miRNA Isolation Kit (AM1561, Life Technologies, USA). .. Then, the differential expression of RNA was identified using miRNA microarray by CapitalBio Corporation (CapitalBio, China). ..



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    Qiagen pcr-based mirna microarray platform
    Total RNA was isolated from WT and IL10KO mice BM-FPCs using miRNeasy kit (Qiagen) and fibrosis-associated <t>miRNA</t> platform was used to assess the miRNAs level using Taqman microRNA assay. (A) At basal level fibrosis-associated miRNAs expression was significantly increased in IL10KO BM-FPCs compared to WT BM-FPCs with maximum expression of miRNA21, 145 and 208a (N=6). (B–D) miRNA21/145/208a expression was measured in WT-FPCs treated with IL10 and TGFβ by RT-PCR. IL10 treatment significantly inhibits TGFβ-induced miRNA-21, -145 and 208a expression. **p<0.01 vs BSA, #p<0.05 vs TGFβ. IL10KO mice: IL10 Knockout mice; BM-FPC: Bone marrow fibroblast progenitor cells. (N=6)
    Pcr Based Mirna Microarray Platform, supplied by Qiagen, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/pcr-based+mirna+microarray+platform/pcr+based+mirna+microarray+platform/pmc05736130-291-16-20
    Average 90 stars, based on 1 article reviews
    pcr-based mirna microarray platform - by Bioz Stars, 2026-11
    90/100 stars
    		  Buy from Supplier

    Image Search Results


    Total RNA was isolated from WT and IL10KO mice BM-FPCs using miRNeasy kit (Qiagen) and fibrosis-associated miRNA platform was used to assess the miRNAs level using Taqman microRNA assay. (A) At basal level fibrosis-associated miRNAs expression was significantly increased in IL10KO BM-FPCs compared to WT BM-FPCs with maximum expression of miRNA21, 145 and 208a (N=6). (B–D) miRNA21/145/208a expression was measured in WT-FPCs treated with IL10 and TGFβ by RT-PCR. IL10 treatment significantly inhibits TGFβ-induced miRNA-21, -145 and 208a expression. **p<0.01 vs BSA, #p<0.05 vs TGFβ. IL10KO mice: IL10 Knockout mice; BM-FPC: Bone marrow fibroblast progenitor cells. (N=6)

    Journal: Circulation

    Article Title: Interleukin 10 Inhibits Bone Marrow Fibroblast Progenitor Cell-mediated Cardiac Fibrosis in Pressure Overloaded Myocardium

    doi: 10.1161/CIRCULATIONAHA.117.027889

    Figure Lengend Snippet: Total RNA was isolated from WT and IL10KO mice BM-FPCs using miRNeasy kit (Qiagen) and fibrosis-associated miRNA platform was used to assess the miRNAs level using Taqman microRNA assay. (A) At basal level fibrosis-associated miRNAs expression was significantly increased in IL10KO BM-FPCs compared to WT BM-FPCs with maximum expression of miRNA21, 145 and 208a (N=6). (B–D) miRNA21/145/208a expression was measured in WT-FPCs treated with IL10 and TGFβ by RT-PCR. IL10 treatment significantly inhibits TGFβ-induced miRNA-21, -145 and 208a expression. **p<0.01 vs BSA, #p<0.05 vs TGFβ. IL10KO mice: IL10 Knockout mice; BM-FPC: Bone marrow fibroblast progenitor cells. (N=6)

    Article Snippet: Fibrosis-associated miRNA expression was analyzed in WT and IL10 KO BM-FPCs at basal level using a PCR-based miRNA microarray platform (Qiagen). miRNA-array data showed that several miRNAs were differentially expressed in IL10KO BM-FPCs compared to WT BM-FPCs ( ).

    Techniques: Isolation, TaqMan microRNA Assay, Expressing, Reverse Transcription Polymerase Chain Reaction, Knock-Out

    BM-FPCs were transfected with miRNA-21 mimic for 12 hours using lipofectamine 2000 transfection reagent before IL10 and TGFβ treatments and miRNA-21 expression was measured by RT-PCR. (A) miRNA-21 mimic transfection significantly increased miRNA-21 expression in BM-FPCs. (B–E) 24 hrs after mimic, IL10 and TGFβ treatments, cells were harvested to measured the expression of fibrosis marker genes by RT-PCR. Relative levels of target genes expression were normalized with 18S. IL10 significantly inhibited TGFβ-induced fibrosis-associated genes expression. Restoration of miRNA-21 expression using miRNA-21 mimic significantly attenuated the IL10 effect on expression of these genes. ***p<0.001, **p<0.01, *p<0.05 vs BSA, ###p<0.001, ##p<0.01 vs TGFβ; $$$p<0.001, $p<0.05 vs TGFβ and ^^^p<0.001, ^^p<0.01, ^p<0.05 vs miR-21 mimic alone. (N=6) (F) Schematic diagram indicates TAC-induced mobilization and homing of BM-FPCs to the heart. In the heart, profibrotic stimuli (TGFβ in this case) first facilitates BM-FPC activation to myofibroblasts and then promote Smad2/3 dependent miRNA-21 maturation. The miRNA-21 maturation ultimately leads to fibrotic gene activation in activated myofibroblasts. On the other hand, IL10 inhibits TAC-induced mobilization, homing and activation of these cells and regulates cardiac fibrosis. Factor: Chemokine/cytokines; BMCs: Bone marrow cells; IL10 Interleukin-10.

    Journal: Circulation

    Article Title: Interleukin 10 Inhibits Bone Marrow Fibroblast Progenitor Cell-mediated Cardiac Fibrosis in Pressure Overloaded Myocardium

    doi: 10.1161/CIRCULATIONAHA.117.027889

    Figure Lengend Snippet: BM-FPCs were transfected with miRNA-21 mimic for 12 hours using lipofectamine 2000 transfection reagent before IL10 and TGFβ treatments and miRNA-21 expression was measured by RT-PCR. (A) miRNA-21 mimic transfection significantly increased miRNA-21 expression in BM-FPCs. (B–E) 24 hrs after mimic, IL10 and TGFβ treatments, cells were harvested to measured the expression of fibrosis marker genes by RT-PCR. Relative levels of target genes expression were normalized with 18S. IL10 significantly inhibited TGFβ-induced fibrosis-associated genes expression. Restoration of miRNA-21 expression using miRNA-21 mimic significantly attenuated the IL10 effect on expression of these genes. ***p<0.001, **p<0.01, *p<0.05 vs BSA, ###p<0.001, ##p<0.01 vs TGFβ; $$$p<0.001, $p<0.05 vs TGFβ and ^^^p<0.001, ^^p<0.01, ^p<0.05 vs miR-21 mimic alone. (N=6) (F) Schematic diagram indicates TAC-induced mobilization and homing of BM-FPCs to the heart. In the heart, profibrotic stimuli (TGFβ in this case) first facilitates BM-FPC activation to myofibroblasts and then promote Smad2/3 dependent miRNA-21 maturation. The miRNA-21 maturation ultimately leads to fibrotic gene activation in activated myofibroblasts. On the other hand, IL10 inhibits TAC-induced mobilization, homing and activation of these cells and regulates cardiac fibrosis. Factor: Chemokine/cytokines; BMCs: Bone marrow cells; IL10 Interleukin-10.

    Article Snippet: Fibrosis-associated miRNA expression was analyzed in WT and IL10 KO BM-FPCs at basal level using a PCR-based miRNA microarray platform (Qiagen). miRNA-array data showed that several miRNAs were differentially expressed in IL10KO BM-FPCs compared to WT BM-FPCs ( ).

    Techniques: Transfection, Expressing, Reverse Transcription Polymerase Chain Reaction, Marker, Activation Assay