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nzw j female mice jackson laboratory rrid imsr jax  (Jackson Laboratory)

 
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    Structured Review

    Jackson Laboratory nzw j female mice jackson laboratory rrid imsr jax
    Nzw J Female Mice Jackson Laboratory Rrid Imsr Jax, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/controller/control+mice/pm41275496-319-152-155
    Average 86 stars, based on 1 article reviews
    nzw j female mice jackson laboratory rrid imsr jax - by Bioz Stars, 2026-10
    86/100 stars

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    Related Articles

    Control:

    Article Title: IL-17RC signaling connects intestinal microbiota and neuroimmune interactions in atherosclerosis
    Article Snippet: .. C57BL/6J control (000664), Ldlr −/− (002207), Villin Cre (021504), Th Cre (008601) were purchased from Jackson Laboratories and kept in our facility. ll17rc flox/flox ( ll17rc flox/flox were generated in the lab in collaboration with Dr. Grivennikov ( and ). ll17rc flox/flox were bred with Ldlr −/− , Villin Cre or Th cre mice to obtain Ldlr −/− x ll17rc flox/flox x Villin Cre (referred in text as Ldlr −/− ll17rc Δ IEC ) and ll17rc flox/flox x Th cre (referred in the text as ll17rc ThCre ) mice. ..

    Article Title: An Advanced Single-Cell RNA Sequencing (scRNA-seq) Protocol Utilizing Custom-Designed Multiplexing
    Article Snippet: .. Wild-type (WT) control mice were purchased from The Jackson Laboratory (Bar Harbor, ME). ..

    Article Title: Early Development of Ocadusertib, a Selective Receptor‐Interacting Serine/Threonine‐Protein Kinase 1 Inhibitor
    Article Snippet: .. Female and male Sharpin cpdm and littermate control mice (~5 weeks old) were purchased from Jackson Laboratories and assigned to groups ( n = 14–20/group). ..

    Article Title: Loss of LanC-like proteins impairs post-injury regeneration of aging muscles
    Article Snippet: .. LanCL1/2/3 triple knockout (LTKO) mice [ ] and wild-type (WT) control mice were maintained on an FVB/NJ background (Jackson Laboratory, stock number 001800). ..

    Article Title: Linking cross-species trajectories of cerebrovascular remodeling in aging and Alzheimer’s disease to brain vessel transcriptome
    Article Snippet: We used B6.Cg-Tg(APPswe,PSEN1dE9)85Dbo mice, male and female (Jackson Labs-MMRC) transgenic for chimeric mouse/human amyloid precursor protein (Mo/HuAPP695swe) and mutant human presenilin 1 (PS1-dE9) directed to neurons ( ). .. Control mice (B6C3, Jackson Labs) from the background of the AD model were age and sex matched (N = 9 AD male, 9 AD female, 9 control male, 9 control female for total of 36 mice across all groups). ..

    Article Title: Acetylcholinesterase inhibitor therapy mitigates hypertension in lupus mice
    Article Snippet: .. Female SLE ( NZBWF1 ) and control ( NZW/LacJ ) mice were obtained from Jackson Laboratories (Bar Harbor, ME) at 3–6 weeks of age. ..

    Article Title: Extensive transcriptomic changes in cellular and animal models of Huntington's disease depending on the length of CAG repeats in the exon 1 of the HTT gene.
    Article Snippet: Although Huntington's disease – a severe, inherited, neurodegenerative disorder – is primarily caused by a pathological variant of the HTT gene (encoding huntingtin protein) which is characterized by the extension of CAG repeats in the 1st exon exceeding 36 triplets, the biochemical mechanisms underlying the disease remain poorly understood.. Mutant huntingtin forms toxic aggregates in cells; however, the clinical symptoms usually appear in adulthood.. Recent reports suggested that somatic expansion of CAG repeats to more than 150 copies may be responsible for the pathogenicity and could explain the delayed onset of symptoms in this inherited disease.

    Article Title: Gasdermin E is dispensable for H1N1 influenza virus pathogenesis in mice
    Article Snippet: .. Male and female Gsdme -/- mice (Jackson Laboratory strain #032411) ( ) and C57BL/6NJ WT control mice (Jackson Laboratory strain #005304), ages 7–10 weeks, were used for all experiments. ..

    Generated:

    Article Title: IL-17RC signaling connects intestinal microbiota and neuroimmune interactions in atherosclerosis
    Article Snippet: .. C57BL/6J control (000664), Ldlr −/− (002207), Villin Cre (021504), Th Cre (008601) were purchased from Jackson Laboratories and kept in our facility. ll17rc flox/flox ( ll17rc flox/flox were generated in the lab in collaboration with Dr. Grivennikov ( and ). ll17rc flox/flox were bred with Ldlr −/− , Villin Cre or Th cre mice to obtain Ldlr −/− x ll17rc flox/flox x Villin Cre (referred in text as Ldlr −/− ll17rc Δ IEC ) and ll17rc flox/flox x Th cre (referred in the text as ll17rc ThCre ) mice. ..

    Triple Knockout:

    Article Title: Loss of LanC-like proteins impairs post-injury regeneration of aging muscles
    Article Snippet: .. LanCL1/2/3 triple knockout (LTKO) mice [ ] and wild-type (WT) control mice were maintained on an FVB/NJ background (Jackson Laboratory, stock number 001800). ..

    Mouse Assay:

    Article Title: Loss of LanC-like proteins impairs post-injury regeneration of aging muscles
    Article Snippet: .. LanCL1/2/3 triple knockout (LTKO) mice [ ] and wild-type (WT) control mice were maintained on an FVB/NJ background (Jackson Laboratory, stock number 001800). ..

    Transgenic Assay:

    Article Title: Extensive transcriptomic changes in cellular and animal models of Huntington's disease depending on the length of CAG repeats in the exon 1 of the HTT gene.
    Article Snippet: Although Huntington's disease – a severe, inherited, neurodegenerative disorder – is primarily caused by a pathological variant of the HTT gene (encoding huntingtin protein) which is characterized by the extension of CAG repeats in the 1st exon exceeding 36 triplets, the biochemical mechanisms underlying the disease remain poorly understood.. Mutant huntingtin forms toxic aggregates in cells; however, the clinical symptoms usually appear in adulthood.. Recent reports suggested that somatic expansion of CAG repeats to more than 150 copies may be responsible for the pathogenicity and could explain the delayed onset of symptoms in this inherited disease.



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