axio observer inverted microscope with incubation (Carl Zeiss)
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Carl Zeiss
axio observer inverted microscope with incubation
Axio Observer Inverted Microscope With Incubation, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 96/100, based on 292 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/Inverted+Microscopes/Inverted+microscope+Axio+Observer+5/pm41589350-92-8-25
Average 96 stars, based on 292 article reviews
Axio Observer Inverted Microscope With Incubation, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 96/100, based on 292 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/Inverted+Microscopes/Inverted+microscope+Axio+Observer+5/pm41589350-92-8-25
Average 96 stars, based on 292 article reviews
axio observer inverted microscope with incubation - by Bioz Stars,
2026-10
96/100 stars
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Inverted Microscopy:Article Title: Galectin-1/ galectin-3 chimeras and multivalent proteins Article Snippet: .. Images were taken on a Zeiss Microscopy:Article Title: Phosphorylation-driven Targeted Protein Degradation of Oncogenic β-catenin Article Snippet: Finally, cover glasses were mounted on microscope slides with Fluoromount Aqueous Mounting Medium (Sigma-Aldrich, F4680). .. Images were acquired in the .czi format using a motorised inverted Article Title: Ca 2+ and DRP1 drive endocytic lysosome reformation at tripartite contact sites Article Snippet: .. Live-cell imaging was performed at 37°C in 5% CO 2 using an inverted Article Title: Cytoplasmic-nuclear remote suppression of c-MYC induces cell cycle arrest and enhances radiotherapy sensitivity in liver cancer via biohybrid platelets Article Snippet: Radiotherapy resistance in liver cancer is intrinsically linked to nuclear DNA repair pathways, yet directly targeting intranuclear regulators remains hindered by biological barriers.. This study pioneered a cytoplasm-tonucleus regulatory strategy by exploiting tripartite motif-containing protein 31 (TRIM31), a cytoplasmic E3 ligase that controlled nuclear c-MYC through the ROS-Wnt/β-catenin axis, bypassing the need for direct nuclear drug delivery.. TRIM31 knockdown triggered a redox storm, which impaired β-catenin nuclear translocation, thereby suppressing c-MYC-driven DNA repair through enforced G1/S phase arrest. Article Title: Ca 2+ and DRP1 drive endocytic lysosome reformation at tripartite contact sites Article Snippet: .. Images were acquired at 37°C with 5% CO 2 using an inverted Software:Article Title: Phosphorylation-driven Targeted Protein Degradation of Oncogenic β-catenin Article Snippet: Finally, cover glasses were mounted on microscope slides with Fluoromount Aqueous Mounting Medium (Sigma-Aldrich, F4680). .. Images were acquired in the .czi format using a motorised inverted Article Title: Ca 2+ and DRP1 drive endocytic lysosome reformation at tripartite contact sites Article Snippet: .. Live-cell imaging was performed at 37°C in 5% CO 2 using an inverted Article Title: Ca 2+ and DRP1 drive endocytic lysosome reformation at tripartite contact sites Article Snippet: .. Images were acquired at 37°C with 5% CO 2 using an inverted Live Cell Imaging:Article Title: Ca 2+ and DRP1 drive endocytic lysosome reformation at tripartite contact sites Article Snippet: .. Live-cell imaging was performed at 37°C in 5% CO 2 using an inverted other:Article Title: Saquinavir induces pyroptosis through the OTUD5-JAK1-GSDME axis in hepatocellular carcinoma. Article Snippet: Pyroptosis is a newly defined form of programmed cell death characterized by plasma membrane perforation, release of cellular contents, and a robust inflammatory response, thereby sensitizing tumors to existing anticancer therapies.. Our study aimed to screen a panel of clinically used antiviral drugs to identify candidate compounds capable of inducing pyroptosis in hepatocellular carcinoma (HCC) cells by evaluating cell viability and monitoring morphological changes.. In vitro experiments using two HCC cell lines and xenograft models were established to investigate the mechanism and combined therapeutic efficacy of the candidate drugs. Article Title: Apoptotic metabolites synthesize and inherit unique de novo L2a/L2b RNAs to prevent virus infection Article Snippet: The osteogenic nodules and lipid droplets were observed under the Article Title: Lensless Quantitative Phase Imaging with Bayer-Filtered Color Sensors Under Sequential RGB-LED Illumination Article Snippet: For reference, corresponding fields of view were acquired using a commercial Staining:Article Title: Cytoplasmic-nuclear remote suppression of c-MYC induces cell cycle arrest and enhances radiotherapy sensitivity in liver cancer via biohybrid platelets Article Snippet: Radiotherapy resistance in liver cancer is intrinsically linked to nuclear DNA repair pathways, yet directly targeting intranuclear regulators remains hindered by biological barriers.. This study pioneered a cytoplasm-tonucleus regulatory strategy by exploiting tripartite motif-containing protein 31 (TRIM31), a cytoplasmic E3 ligase that controlled nuclear c-MYC through the ROS-Wnt/β-catenin axis, bypassing the need for direct nuclear drug delivery.. TRIM31 knockdown triggered a redox storm, which impaired β-catenin nuclear translocation, thereby suppressing c-MYC-driven DNA repair through enforced G1/S phase arrest. Fluorescence:Article Title: Cytoplasmic-nuclear remote suppression of c-MYC induces cell cycle arrest and enhances radiotherapy sensitivity in liver cancer via biohybrid platelets Article Snippet: Radiotherapy resistance in liver cancer is intrinsically linked to nuclear DNA repair pathways, yet directly targeting intranuclear regulators remains hindered by biological barriers.. This study pioneered a cytoplasm-tonucleus regulatory strategy by exploiting tripartite motif-containing protein 31 (TRIM31), a cytoplasmic E3 ligase that controlled nuclear c-MYC through the ROS-Wnt/β-catenin axis, bypassing the need for direct nuclear drug delivery.. TRIM31 knockdown triggered a redox storm, which impaired β-catenin nuclear translocation, thereby suppressing c-MYC-driven DNA repair through enforced G1/S phase arrest. |