psb1115 Search Results


psb1115  (MedChemExpress)
94
MedChemExpress psb1115
Psb1115, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/psb1115/PSB-1115/pm41957361-317-0-2
Average 94 stars, based on 1 article reviews
psb1115 - by Bioz Stars, 2026-10
94/100 stars
  Buy from Supplier
psb 1115  (TargetMol)
N/A
   Buy from Supplier
psb1115  (Tocris)
94
Tocris psb1115
Psb1115, supplied by Tocris, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/psb1115/PSB+1115/pmc04838392-231-11-16
Average 94 stars, based on 1 article reviews
psb1115 - by Bioz Stars, 2026-10
94/100 stars
  Buy from Supplier
a2bar antagonist psb1115  (Santa Cruz Biotechnology)
90
Santa Cruz Biotechnology a2bar antagonist psb1115
A2bar Antagonist Psb1115, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/psb1115/PSB+1115/pmc07253199-193-8-11
Average 90 stars, based on 1 article reviews
a2bar antagonist psb1115 - by Bioz Stars, 2026-10
90/100 stars
  Buy from Supplier
psb 1115 potassium salt  (Tocris)
93
Tocris psb 1115 potassium salt
Psb 1115 Potassium Salt, supplied by Tocris, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/psb1115/PSB+1115/pm38920052__cb4c00210_si_001-102-0-7
Average 93 stars, based on 1 article reviews
psb 1115 potassium salt - by Bioz Stars, 2026-10
93/100 stars
  Buy from Supplier
88 psb1115  (ApexBio)
90
ApexBio 88 psb1115
88 Psb1115, supplied by ApexBio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/psb1115/88+psb1115/pm32129084-34-7-14
Average 90 stars, based on 1 article reviews
88 psb1115 - by Bioz Stars, 2026-10
90/100 stars
  Buy from Supplier
psb1115  (GlpBio Technology Inc)
90
GlpBio Technology Inc psb1115
The apoptosis and migration of HP-infected GES-1 cells were facilitated by A2BR activation. A. The morphology of GES-1 cells was observed under an inverted microscope. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM <t>PSB1115.</t> B. Flow cytometry was utilized to measure the apoptotic rate. C. The transwell assay was used to evaluate the migration ability. D. The metastasis of cells was determined using the wound healing assay (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).
Psb1115, supplied by GlpBio Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/psb1115/psb1115/pmc10660004-40-53-54
Average 90 stars, based on 1 article reviews
psb1115 - by Bioz Stars, 2026-10
90/100 stars
  Buy from Supplier
psb 1115  (Macklin Inc)
86
Macklin Inc psb 1115
The apoptosis and migration of HP-infected GES-1 cells were facilitated by A2BR activation. A. The morphology of GES-1 cells was observed under an inverted microscope. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM <t>PSB1115.</t> B. Flow cytometry was utilized to measure the apoptotic rate. C. The transwell assay was used to evaluate the migration ability. D. The metastasis of cells was determined using the wound healing assay (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).
Psb 1115, supplied by Macklin Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/psb1115/1115+psb/pmc12607671-233-28-29
Average 86 stars, based on 1 article reviews
psb 1115 - by Bioz Stars, 2026-10
86/100 stars
  Buy from Supplier
psb 1115  (BOC Sciences)
N/A
Selective human A2B receptor antagonist
   Buy from Supplier
psb 1115  (Cayman Chemical)
N/A
PSB 1115 is an antagonist of the adenosine A receptor K 53 4 nM It is selective for A over A and A receptors which have K values of 2 200 and 24 000 nM
   Buy from Supplier
psb-1115 potassium salt  (TargetMol)
N/A
   Buy from Supplier
psb 1115  (Toronto Research Chemicals)
N/A
   Buy from Supplier

Image Search Results


The apoptosis and migration of HP-infected GES-1 cells were facilitated by A2BR activation. A. The morphology of GES-1 cells was observed under an inverted microscope. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM PSB1115. B. Flow cytometry was utilized to measure the apoptotic rate. C. The transwell assay was used to evaluate the migration ability. D. The metastasis of cells was determined using the wound healing assay (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Journal: Heliyon

Article Title: A2BR facilitates the pathogenesis of H. pylori-associated GU by inducing oxidative stress through p38 MAPK phosphorylation

doi: 10.1016/j.heliyon.2023.e21004

Figure Lengend Snippet: The apoptosis and migration of HP-infected GES-1 cells were facilitated by A2BR activation. A. The morphology of GES-1 cells was observed under an inverted microscope. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM PSB1115. B. Flow cytometry was utilized to measure the apoptotic rate. C. The transwell assay was used to evaluate the migration ability. D. The metastasis of cells was determined using the wound healing assay (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Article Snippet: In the HP group, GES-1 cells were infected with HP for 24 h. In the HP + BAY60-6583 group, HP-infected GES-1 cells were incubated with 10 μM BAY60-6583 (MCE, USA), an agonist of A2BR [ ], for 24 h. In the HP + PSB1115 group, HP-infected GES-1 cells were incubated with 25 nM PSB1115 (GlpBio, USA), an antagonist of A2BR [ ], for 24 h. Apoptosis analysis: Cells were seeded in 6-well plates and incubated with Annexin V-fluorescein isothiocyanate (556,547; BD Biosciences, USA), followed by the addition of propidium iodide.

Techniques: Migration, Infection, Activation Assay, Inverted Microscopy, Incubation, Flow Cytometry, Transwell Assay, Wound Healing Assay, Control

Oxidative stress in HP-infected GES-1 cells was aggravated by A2BR activation. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM PSB1115. A. The ROS level in GES-1 cells was evaluated by the DCFH-DA assay. B. The MDA level was determined by a commercial kit using the TBA method. C. SOD activity was checked by a commercial kit using the WST-1 method (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Journal: Heliyon

Article Title: A2BR facilitates the pathogenesis of H. pylori-associated GU by inducing oxidative stress through p38 MAPK phosphorylation

doi: 10.1016/j.heliyon.2023.e21004

Figure Lengend Snippet: Oxidative stress in HP-infected GES-1 cells was aggravated by A2BR activation. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM PSB1115. A. The ROS level in GES-1 cells was evaluated by the DCFH-DA assay. B. The MDA level was determined by a commercial kit using the TBA method. C. SOD activity was checked by a commercial kit using the WST-1 method (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Article Snippet: In the HP group, GES-1 cells were infected with HP for 24 h. In the HP + BAY60-6583 group, HP-infected GES-1 cells were incubated with 10 μM BAY60-6583 (MCE, USA), an agonist of A2BR [ ], for 24 h. In the HP + PSB1115 group, HP-infected GES-1 cells were incubated with 25 nM PSB1115 (GlpBio, USA), an antagonist of A2BR [ ], for 24 h. Apoptosis analysis: Cells were seeded in 6-well plates and incubated with Annexin V-fluorescein isothiocyanate (556,547; BD Biosciences, USA), followed by the addition of propidium iodide.

Techniques: Infection, Activation Assay, Incubation, DCFH-DA Assay, Activity Assay, Control

P38MAPK signaling in HP-infected GES-1 cells was activated by A2BR activation. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM PSB1115. The expression levels of p38 and p-p38 were evaluated by Western blotting (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Journal: Heliyon

Article Title: A2BR facilitates the pathogenesis of H. pylori-associated GU by inducing oxidative stress through p38 MAPK phosphorylation

doi: 10.1016/j.heliyon.2023.e21004

Figure Lengend Snippet: P38MAPK signaling in HP-infected GES-1 cells was activated by A2BR activation. GES-1 cells were infected with HP, followed by incubation with 10 μM BAY60-6583 and 25 nM PSB1115. The expression levels of p38 and p-p38 were evaluated by Western blotting (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Article Snippet: In the HP group, GES-1 cells were infected with HP for 24 h. In the HP + BAY60-6583 group, HP-infected GES-1 cells were incubated with 10 μM BAY60-6583 (MCE, USA), an agonist of A2BR [ ], for 24 h. In the HP + PSB1115 group, HP-infected GES-1 cells were incubated with 25 nM PSB1115 (GlpBio, USA), an antagonist of A2BR [ ], for 24 h. Apoptosis analysis: Cells were seeded in 6-well plates and incubated with Annexin V-fluorescein isothiocyanate (556,547; BD Biosciences, USA), followed by the addition of propidium iodide.

Techniques: Infection, Activation Assay, Incubation, Expressing, Western Blot, Control

The pathological state of gastric ulcers in HP-treated rats was aggravated by A2BR activation. Rats were treated with 2 mg/kg BAY60-6583 or 10 mg/kg PSB1115, followed by gastric ulcer modeling 30 min later. A. The ulcer area in each group was calculated. B. The pepsin activity in each group was determined using the stop-point assay of denatured hemoglobin hydrolysis. C. The pathological changes in gastric mucosa tissues were checked by the HE staining assay. D. The MDA level in gastric mucosa tissues was determined by a commercial kit using the TBA method. E. The SOD activity in gastric mucosa tissues was checked by a commercial kit using the WST-1 method (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Journal: Heliyon

Article Title: A2BR facilitates the pathogenesis of H. pylori-associated GU by inducing oxidative stress through p38 MAPK phosphorylation

doi: 10.1016/j.heliyon.2023.e21004

Figure Lengend Snippet: The pathological state of gastric ulcers in HP-treated rats was aggravated by A2BR activation. Rats were treated with 2 mg/kg BAY60-6583 or 10 mg/kg PSB1115, followed by gastric ulcer modeling 30 min later. A. The ulcer area in each group was calculated. B. The pepsin activity in each group was determined using the stop-point assay of denatured hemoglobin hydrolysis. C. The pathological changes in gastric mucosa tissues were checked by the HE staining assay. D. The MDA level in gastric mucosa tissues was determined by a commercial kit using the TBA method. E. The SOD activity in gastric mucosa tissues was checked by a commercial kit using the WST-1 method (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Article Snippet: In the HP group, GES-1 cells were infected with HP for 24 h. In the HP + BAY60-6583 group, HP-infected GES-1 cells were incubated with 10 μM BAY60-6583 (MCE, USA), an agonist of A2BR [ ], for 24 h. In the HP + PSB1115 group, HP-infected GES-1 cells were incubated with 25 nM PSB1115 (GlpBio, USA), an antagonist of A2BR [ ], for 24 h. Apoptosis analysis: Cells were seeded in 6-well plates and incubated with Annexin V-fluorescein isothiocyanate (556,547; BD Biosciences, USA), followed by the addition of propidium iodide.

Techniques: Activation Assay, Activity Assay, Staining, Control

P38MAPK signaling in HP-treated rats was activated by A2BR activation. Rats were treated with 2 mg/kg BAY60-6583 or 10 mg/kg PSB1115, followed by gastric ulcer modeling 30 min later. The expression levels of p38 and p-p38 in gastric mucosal tissues were evaluated by Western blotting (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Journal: Heliyon

Article Title: A2BR facilitates the pathogenesis of H. pylori-associated GU by inducing oxidative stress through p38 MAPK phosphorylation

doi: 10.1016/j.heliyon.2023.e21004

Figure Lengend Snippet: P38MAPK signaling in HP-treated rats was activated by A2BR activation. Rats were treated with 2 mg/kg BAY60-6583 or 10 mg/kg PSB1115, followed by gastric ulcer modeling 30 min later. The expression levels of p38 and p-p38 in gastric mucosal tissues were evaluated by Western blotting (**p < 0.01 vs. control, #p < 0.05 vs. HP, ##p < 0.01 vs. HP).

Article Snippet: In the HP group, GES-1 cells were infected with HP for 24 h. In the HP + BAY60-6583 group, HP-infected GES-1 cells were incubated with 10 μM BAY60-6583 (MCE, USA), an agonist of A2BR [ ], for 24 h. In the HP + PSB1115 group, HP-infected GES-1 cells were incubated with 25 nM PSB1115 (GlpBio, USA), an antagonist of A2BR [ ], for 24 h. Apoptosis analysis: Cells were seeded in 6-well plates and incubated with Annexin V-fluorescein isothiocyanate (556,547; BD Biosciences, USA), followed by the addition of propidium iodide.

Techniques: Activation Assay, Expressing, Western Blot, Control