nbqx Search Results


2 3 dioxo 6 nitro 1 2 3 4tetrahydrobenzo  (Tocris)
96
Tocris 2 3 dioxo 6 nitro 1 2 3 4tetrahydrobenzo
2 3 Dioxo 6 Nitro 1 2 3 4tetrahydrobenzo, supplied by Tocris, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/2 3 dioxo 6 nitro 1 2 3 4tetrahydrobenzo/product/Tocris
Average 96 stars, based on 1 article reviews
2 3 dioxo 6 nitro 1 2 3 4tetrahydrobenzo - by Bioz Stars, 2026-03
96/100 stars
  Buy from Supplier
nbqx  (Alomone Labs)
93
Alomone Labs nbqx
Nbqx, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx/product/Alomone Labs
Average 93 stars, based on 1 article reviews
nbqx - by Bioz Stars, 2026-03
93/100 stars
  Buy from Supplier
2 3 dihydroxy 6 nitro 7 sulfonyl benzo  (Tocris)
97
Tocris 2 3 dihydroxy 6 nitro 7 sulfonyl benzo
2 3 Dihydroxy 6 Nitro 7 Sulfonyl Benzo, supplied by Tocris, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/2 3 dihydroxy 6 nitro 7 sulfonyl benzo/product/Tocris
Average 97 stars, based on 1 article reviews
2 3 dihydroxy 6 nitro 7 sulfonyl benzo - by Bioz Stars, 2026-03
97/100 stars
  Buy from Supplier
nbqx disodium salt  (Tocris)
96
Tocris nbqx disodium salt
Nbqx Disodium Salt, supplied by Tocris, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx disodium salt/product/Tocris
Average 96 stars, based on 1 article reviews
nbqx disodium salt - by Bioz Stars, 2026-03
96/100 stars
  Buy from Supplier
n-186  (alomone labs)
92
alomone labs n-186
N 186, supplied by alomone labs, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/n-186/product/alomone labs
Average 92 stars, based on 1 article reviews
n-186 - by Bioz Stars, 2026-03
92/100 stars
  Buy from Supplier
nbqx  (MedChemExpress)
93
MedChemExpress nbqx
Optogenetically activated astrocytes induced excitatory response in SF-1 neurons, mainly via NMDA receptor. (a) Schematic representing viral construct of ChR2 specifically expressed in astrocytes. (b) Whole-cell recording of VMH neurons near ChR2-expressing astrocytes. (c) Representative recordings illustrating VMH SF-1 neurons depolarized by optical stimulation of astrocytes. (d) Application of glutamatergic receptor antagonists <t>NBQX</t> (50 μ M) <t>and</t> <t>D-APV</t> (50 μ M) suppressed action potential firing caused by optical stimulation of astrocytes, which was recovered after aCSF washout ( n = 6). (e) Application of D-APV significantly diminished membrane potential depolarization induced by astrocyte activation ( n = 5, ∗∗ p < 0.01; paired t -test). (f) NBQX did not influence interaction between astrocytes and SF-1 neurons ( n = 5, ∗ p < 0.05; paired t -test). (g) Schematic showing that manipulation of astrocytes in VMH could bidirectionally regulate chronic stress-induced anxiety and bone loss by affecting neural excitatory through NMDA receptors.
Nbqx, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx/product/MedChemExpress
Average 93 stars, based on 1 article reviews
nbqx - by Bioz Stars, 2026-03
93/100 stars
  Buy from Supplier
nbqx disodium salt  (TargetMol)
93
TargetMol nbqx disodium salt
Optogenetically activated astrocytes induced excitatory response in SF-1 neurons, mainly via NMDA receptor. (a) Schematic representing viral construct of ChR2 specifically expressed in astrocytes. (b) Whole-cell recording of VMH neurons near ChR2-expressing astrocytes. (c) Representative recordings illustrating VMH SF-1 neurons depolarized by optical stimulation of astrocytes. (d) Application of glutamatergic receptor antagonists <t>NBQX</t> (50 μ M) <t>and</t> <t>D-APV</t> (50 μ M) suppressed action potential firing caused by optical stimulation of astrocytes, which was recovered after aCSF washout ( n = 6). (e) Application of D-APV significantly diminished membrane potential depolarization induced by astrocyte activation ( n = 5, ∗∗ p < 0.01; paired t -test). (f) NBQX did not influence interaction between astrocytes and SF-1 neurons ( n = 5, ∗ p < 0.05; paired t -test). (g) Schematic showing that manipulation of astrocytes in VMH could bidirectionally regulate chronic stress-induced anxiety and bone loss by affecting neural excitatory through NMDA receptors.
Nbqx Disodium Salt, supplied by TargetMol, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx disodium salt/product/TargetMol
Average 93 stars, based on 1 article reviews
nbqx disodium salt - by Bioz Stars, 2026-03
93/100 stars
  Buy from Supplier
recombinant proteins nbqx  (Toronto Research Chemicals)
95
Toronto Research Chemicals recombinant proteins nbqx
(A) Typical electrode placement for synaptic stimulation. The picture shows an infra-red differential interference contrast image of a horizontal slice from the hippocampus, where the stimulating electrode (s.e.) is placed in the molecular layer (m.l.) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (B) Inhibition of ionotropic glutamate receptors abolishes the metabolic transients. A representative trace of a synaptically stimulated DGN (using a train of 60 pulses at 20 Hz) shows that the Peredox signal disappears after blocking synaptic transmission with a mixture of 5 μM <t>NBQX</t> and 25 <t>μM</t> <t>D-AP5</t> in the ACSF. The complete blockade of synaptic transmission is corroborated by the absence of the typical RCaMP1h signal after stimulation. The effect of synaptic blockers was partially reversed after 5 min of washout in regular ACSF, and both Ca2+ and NADH transients were completely recovered after 10 min in ACSF. (C) Dataset for the effect of ionotropic glutamate receptor block. Data were compared by a paired Student’s t-test (neurons=46, slices=6, mice=3). (D) Typical electrode placement for antidromic (axonal) stimulation. In this experimental paradigm, the stimulating electrode (s.e.) is placed in the the hilus (hil) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (E) Average transient increases in Peredox lifetime after antidromic stimulation of 100 brief pulses delivered at 50 Hz. Traces represent the mean ± SEM (events=179, neurons=84, slices=18, mice=11; SEM calculated using number of neurons). The lower panel shows the putative average Ca2+ spike in the cells, as a proxy for neuronal excitation. (F) Elevation in the NADH/NAD+ ratio is directly correlated with the neuronal Ca2+ increase after electrical antidromic stimulation in DGNs, similarly to synaptic stimulation. Scatter plots of changes in Peredox lifetime versus variations in RCaMP lifetime for events recorded after antidromic stimulation (open circles; events=205, neurons=87, slices=19, mice=12), overlap with data collected using synaptic stimulation (dark squares).
Recombinant Proteins Nbqx, supplied by Toronto Research Chemicals, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/recombinant proteins nbqx/product/Toronto Research Chemicals
Average 95 stars, based on 1 article reviews
recombinant proteins nbqx - by Bioz Stars, 2026-03
95/100 stars
  Buy from Supplier
nbqx disodium salt nbqx  (Tocris)
96
Tocris nbqx disodium salt nbqx
(A) Typical electrode placement for synaptic stimulation. The picture shows an infra-red differential interference contrast image of a horizontal slice from the hippocampus, where the stimulating electrode (s.e.) is placed in the molecular layer (m.l.) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (B) Inhibition of ionotropic glutamate receptors abolishes the metabolic transients. A representative trace of a synaptically stimulated DGN (using a train of 60 pulses at 20 Hz) shows that the Peredox signal disappears after blocking synaptic transmission with a mixture of 5 μM <t>NBQX</t> and 25 <t>μM</t> <t>D-AP5</t> in the ACSF. The complete blockade of synaptic transmission is corroborated by the absence of the typical RCaMP1h signal after stimulation. The effect of synaptic blockers was partially reversed after 5 min of washout in regular ACSF, and both Ca2+ and NADH transients were completely recovered after 10 min in ACSF. (C) Dataset for the effect of ionotropic glutamate receptor block. Data were compared by a paired Student’s t-test (neurons=46, slices=6, mice=3). (D) Typical electrode placement for antidromic (axonal) stimulation. In this experimental paradigm, the stimulating electrode (s.e.) is placed in the the hilus (hil) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (E) Average transient increases in Peredox lifetime after antidromic stimulation of 100 brief pulses delivered at 50 Hz. Traces represent the mean ± SEM (events=179, neurons=84, slices=18, mice=11; SEM calculated using number of neurons). The lower panel shows the putative average Ca2+ spike in the cells, as a proxy for neuronal excitation. (F) Elevation in the NADH/NAD+ ratio is directly correlated with the neuronal Ca2+ increase after electrical antidromic stimulation in DGNs, similarly to synaptic stimulation. Scatter plots of changes in Peredox lifetime versus variations in RCaMP lifetime for events recorded after antidromic stimulation (open circles; events=205, neurons=87, slices=19, mice=12), overlap with data collected using synaptic stimulation (dark squares).
Nbqx Disodium Salt Nbqx, supplied by Tocris, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx disodium salt nbqx/product/Tocris
Average 96 stars, based on 1 article reviews
nbqx disodium salt nbqx - by Bioz Stars, 2026-03
96/100 stars
  Buy from Supplier
nbqx  (Santa Cruz Biotechnology)
91
Santa Cruz Biotechnology nbqx

Nbqx, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx/product/Santa Cruz Biotechnology
Average 91 stars, based on 1 article reviews
nbqx - by Bioz Stars, 2026-03
91/100 stars
  Buy from Supplier
nbqx  (Biosynth Carbosynth)
90
Biosynth Carbosynth nbqx

Nbqx, supplied by Biosynth Carbosynth, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx/product/Biosynth Carbosynth
Average 90 stars, based on 1 article reviews
nbqx - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier
nbqx  (Biotrend Chemicals)
90
Biotrend Chemicals nbqx

Nbqx, supplied by Biotrend Chemicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/nbqx/product/Biotrend Chemicals
Average 90 stars, based on 1 article reviews
nbqx - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

Image Search Results


Optogenetically activated astrocytes induced excitatory response in SF-1 neurons, mainly via NMDA receptor. (a) Schematic representing viral construct of ChR2 specifically expressed in astrocytes. (b) Whole-cell recording of VMH neurons near ChR2-expressing astrocytes. (c) Representative recordings illustrating VMH SF-1 neurons depolarized by optical stimulation of astrocytes. (d) Application of glutamatergic receptor antagonists NBQX (50 μ M) and D-APV (50 μ M) suppressed action potential firing caused by optical stimulation of astrocytes, which was recovered after aCSF washout ( n = 6). (e) Application of D-APV significantly diminished membrane potential depolarization induced by astrocyte activation ( n = 5, ∗∗ p < 0.01; paired t -test). (f) NBQX did not influence interaction between astrocytes and SF-1 neurons ( n = 5, ∗ p < 0.05; paired t -test). (g) Schematic showing that manipulation of astrocytes in VMH could bidirectionally regulate chronic stress-induced anxiety and bone loss by affecting neural excitatory through NMDA receptors.

Journal: Neural Plasticity

Article Title: Astrocytes in the Ventromedial Hypothalamus Involve Chronic Stress-Induced Anxiety and Bone Loss in Mice

doi: 10.1155/2021/7806370

Figure Lengend Snippet: Optogenetically activated astrocytes induced excitatory response in SF-1 neurons, mainly via NMDA receptor. (a) Schematic representing viral construct of ChR2 specifically expressed in astrocytes. (b) Whole-cell recording of VMH neurons near ChR2-expressing astrocytes. (c) Representative recordings illustrating VMH SF-1 neurons depolarized by optical stimulation of astrocytes. (d) Application of glutamatergic receptor antagonists NBQX (50 μ M) and D-APV (50 μ M) suppressed action potential firing caused by optical stimulation of astrocytes, which was recovered after aCSF washout ( n = 6). (e) Application of D-APV significantly diminished membrane potential depolarization induced by astrocyte activation ( n = 5, ∗∗ p < 0.01; paired t -test). (f) NBQX did not influence interaction between astrocytes and SF-1 neurons ( n = 5, ∗ p < 0.05; paired t -test). (g) Schematic showing that manipulation of astrocytes in VMH could bidirectionally regulate chronic stress-induced anxiety and bone loss by affecting neural excitatory through NMDA receptors.

Article Snippet: NBQX (50 μ M, MCE, Shanghai, China) and D-APV (50 μ M, MCE Shanghai, China) were used in perfused aCSF to block the function of AMPA and NMDA receptors, respectively.

Techniques: Construct, Expressing, Membrane, Activation Assay

(A) Typical electrode placement for synaptic stimulation. The picture shows an infra-red differential interference contrast image of a horizontal slice from the hippocampus, where the stimulating electrode (s.e.) is placed in the molecular layer (m.l.) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (B) Inhibition of ionotropic glutamate receptors abolishes the metabolic transients. A representative trace of a synaptically stimulated DGN (using a train of 60 pulses at 20 Hz) shows that the Peredox signal disappears after blocking synaptic transmission with a mixture of 5 μM NBQX and 25 μM D-AP5 in the ACSF. The complete blockade of synaptic transmission is corroborated by the absence of the typical RCaMP1h signal after stimulation. The effect of synaptic blockers was partially reversed after 5 min of washout in regular ACSF, and both Ca2+ and NADH transients were completely recovered after 10 min in ACSF. (C) Dataset for the effect of ionotropic glutamate receptor block. Data were compared by a paired Student’s t-test (neurons=46, slices=6, mice=3). (D) Typical electrode placement for antidromic (axonal) stimulation. In this experimental paradigm, the stimulating electrode (s.e.) is placed in the the hilus (hil) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (E) Average transient increases in Peredox lifetime after antidromic stimulation of 100 brief pulses delivered at 50 Hz. Traces represent the mean ± SEM (events=179, neurons=84, slices=18, mice=11; SEM calculated using number of neurons). The lower panel shows the putative average Ca2+ spike in the cells, as a proxy for neuronal excitation. (F) Elevation in the NADH/NAD+ ratio is directly correlated with the neuronal Ca2+ increase after electrical antidromic stimulation in DGNs, similarly to synaptic stimulation. Scatter plots of changes in Peredox lifetime versus variations in RCaMP lifetime for events recorded after antidromic stimulation (open circles; events=205, neurons=87, slices=19, mice=12), overlap with data collected using synaptic stimulation (dark squares).

Journal: Cell metabolism

Article Title: Neuronal stimulation triggers neuronal glycolysis and not lactate uptake

doi: 10.1016/j.cmet.2017.06.021

Figure Lengend Snippet: (A) Typical electrode placement for synaptic stimulation. The picture shows an infra-red differential interference contrast image of a horizontal slice from the hippocampus, where the stimulating electrode (s.e.) is placed in the molecular layer (m.l.) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (B) Inhibition of ionotropic glutamate receptors abolishes the metabolic transients. A representative trace of a synaptically stimulated DGN (using a train of 60 pulses at 20 Hz) shows that the Peredox signal disappears after blocking synaptic transmission with a mixture of 5 μM NBQX and 25 μM D-AP5 in the ACSF. The complete blockade of synaptic transmission is corroborated by the absence of the typical RCaMP1h signal after stimulation. The effect of synaptic blockers was partially reversed after 5 min of washout in regular ACSF, and both Ca2+ and NADH transients were completely recovered after 10 min in ACSF. (C) Dataset for the effect of ionotropic glutamate receptor block. Data were compared by a paired Student’s t-test (neurons=46, slices=6, mice=3). (D) Typical electrode placement for antidromic (axonal) stimulation. In this experimental paradigm, the stimulating electrode (s.e.) is placed in the the hilus (hil) and an extracellular recording electrode (r.e.) is inserted in the layer of dentate granule neurons (DGNs, following the arrow direction). (E) Average transient increases in Peredox lifetime after antidromic stimulation of 100 brief pulses delivered at 50 Hz. Traces represent the mean ± SEM (events=179, neurons=84, slices=18, mice=11; SEM calculated using number of neurons). The lower panel shows the putative average Ca2+ spike in the cells, as a proxy for neuronal excitation. (F) Elevation in the NADH/NAD+ ratio is directly correlated with the neuronal Ca2+ increase after electrical antidromic stimulation in DGNs, similarly to synaptic stimulation. Scatter plots of changes in Peredox lifetime versus variations in RCaMP lifetime for events recorded after antidromic stimulation (open circles; events=205, neurons=87, slices=19, mice=12), overlap with data collected using synaptic stimulation (dark squares).

Article Snippet: KEY RESOURCES TABLE REAGENT or RESOURCE SOURCE IDENTIFIER Antibodies Bacterial and Virus Strains AAV9.Syn.RCaMP1h.WPRE.SV40 Penn Vector Core Discontinued AAV9.Syn.jRCaMP1b-NES.WPRE.SV40 Penn Vector Core Cat#AV-9-PV3852 Biological Samples Chemicals, Peptides, and Recombinant Proteins NBQX (6-Nitro-7-sulfamoylbenzo[f]quinoxaline-2,3-dione, Disodium Salt) Toronto Research Chemicals Cat#N550005; CAS:479347-86-9 D-AP5 (D-(-)-2-Amino-5-phosphonopentanoic acid) Abcam Cat#ab120003; CAS:79055-68-8 AR-C155858 Tocris Bioscience Cat#4960; CAS:496791-37-8 Picrotoxin Sigma-Aldrich Cat#P1675; CAS:124-87-8 Cytochalasin B Tocris Bioscience Cat#5474; CAS:14930-96-2 Cytochalasin D Tocris Bioscience Cat#1233; CAS:22144-77-0 GSK-2837808A Tocris Bioscience Cat#5189; CAS:1445879-21-9 Aminooxyacetate (O-(carboxymethyl)hydroxylamine hemihydrate) Sigma-Aldrich Cat#C13408; CAS:2921-14-4 Sodium L-lactate Sigma-Aldrich Cat#71718; CAS:867-56-1 Sodium pyruvate Sigma-Aldrich Cat#P8574; CAS:113-24-6 Critical Commercial Assays Deposited Data Experimental Models: Cell Lines Experimental Models: Organisms/Strains C57BL/6NCrl mice Charles River RRID:IMSR_CRL:27 Oligonucleotides Recombinant DNA AAV.CAG.Peredox.WPRE.SV40 Mongeon et al., 2016 Addgene #73807 AAV.hSyn.Peredox-NLS.WPRE.SV40 This paper Derived from Addgene #32384 AAV.CAG.SweetieTS.WPRE.SV40 This paper AAV.CAG.Laconic.WPRE.SV40 San Martín et al., 2013 Derived from Addgene #44238 AAV.CAG.pHRed.WPRE.SV40 Tantama et al., 2011 Derived from Addgene #31472 Software and Algorithms GraphPad Prism Version 7 GraphPad Origin 2015 OriginLab ScanImage Pologruto et al., 2003 Other Polymeric microporous filters with 8μm pores and 25 μm thick grid structural support Precision Membranes, Provo, UT N/A Open in a separate window Quantification and Statistical Analyses

Techniques: Inhibition, Blocking Assay, Transmission Assay

Quantification and Statistical Analyses

Journal: Cell metabolism

Article Title: Neuronal stimulation triggers neuronal glycolysis and not lactate uptake

doi: 10.1016/j.cmet.2017.06.021

Figure Lengend Snippet: Quantification and Statistical Analyses

Article Snippet: KEY RESOURCES TABLE REAGENT or RESOURCE SOURCE IDENTIFIER Antibodies Bacterial and Virus Strains AAV9.Syn.RCaMP1h.WPRE.SV40 Penn Vector Core Discontinued AAV9.Syn.jRCaMP1b-NES.WPRE.SV40 Penn Vector Core Cat#AV-9-PV3852 Biological Samples Chemicals, Peptides, and Recombinant Proteins NBQX (6-Nitro-7-sulfamoylbenzo[f]quinoxaline-2,3-dione, Disodium Salt) Toronto Research Chemicals Cat#N550005; CAS:479347-86-9 D-AP5 (D-(-)-2-Amino-5-phosphonopentanoic acid) Abcam Cat#ab120003; CAS:79055-68-8 AR-C155858 Tocris Bioscience Cat#4960; CAS:496791-37-8 Picrotoxin Sigma-Aldrich Cat#P1675; CAS:124-87-8 Cytochalasin B Tocris Bioscience Cat#5474; CAS:14930-96-2 Cytochalasin D Tocris Bioscience Cat#1233; CAS:22144-77-0 GSK-2837808A Tocris Bioscience Cat#5189; CAS:1445879-21-9 Aminooxyacetate (O-(carboxymethyl)hydroxylamine hemihydrate) Sigma-Aldrich Cat#C13408; CAS:2921-14-4 Sodium L-lactate Sigma-Aldrich Cat#71718; CAS:867-56-1 Sodium pyruvate Sigma-Aldrich Cat#P8574; CAS:113-24-6 Critical Commercial Assays Deposited Data Experimental Models: Cell Lines Experimental Models: Organisms/Strains C57BL/6NCrl mice Charles River RRID:IMSR_CRL:27 Oligonucleotides Recombinant DNA AAV.CAG.Peredox.WPRE.SV40 Mongeon et al., 2016 Addgene #73807 AAV.hSyn.Peredox-NLS.WPRE.SV40 This paper Derived from Addgene #32384 AAV.CAG.SweetieTS.WPRE.SV40 This paper AAV.CAG.Laconic.WPRE.SV40 San Martín et al., 2013 Derived from Addgene #44238 AAV.CAG.pHRed.WPRE.SV40 Tantama et al., 2011 Derived from Addgene #31472 Software and Algorithms GraphPad Prism Version 7 GraphPad Origin 2015 OriginLab ScanImage Pologruto et al., 2003 Other Polymeric microporous filters with 8μm pores and 25 μm thick grid structural support Precision Membranes, Provo, UT N/A Open in a separate window Quantification and Statistical Analyses

Techniques: Plasmid Preparation, Recombinant, Derivative Assay, Software

Journal: Neuron

Article Title: Activity-Dependent Plasticity of Axo-axonic Synapses at the Axon Initial Segment

doi: 10.1016/j.neuron.2020.01.037

Figure Lengend Snippet:

Article Snippet: NBQX , Santa Cruz Biotechnology , sc-222048; CAS: 479347-86-9.

Techniques: Virus, Plasmid Preparation, Recombinant, Software, Fluorescence, Imaging