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BioResource International Inc
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INOVOTION
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European Collection of Authenticated Cell Cultures
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Image Search Results
Journal: Asian Pacific Journal of Cancer Prevention : APJCP
Article Title: Terminalia Catappa Extract (TCE) Reduces Proliferation of Lung and Breast Cancer Cell by Modulating miR-21 and miR-34a Expressions
doi: 10.31557/APJCP.2021.22.4.1157
Figure Lengend Snippet: Effect of IC 50 Concentration of TCE on A549, MCF-7, and MDA-231 Cell Proliferation: TCE decreased the cell survival to 17.6%, 21.4%, and 27.21 in 200 µg concentration compared to the control cells
Article Snippet: Cancer Cell Cultures MCF7, MDA 231, and
Techniques: Concentration Assay, Control
Journal: Asian Pacific Journal of Cancer Prevention : APJCP
Article Title: Terminalia Catappa Extract (TCE) Reduces Proliferation of Lung and Breast Cancer Cell by Modulating miR-21 and miR-34a Expressions
doi: 10.31557/APJCP.2021.22.4.1157
Figure Lengend Snippet: Effects of TCE Treatment on the Expression of BAX, Bcl-2, MMP-13, miR-21, miR-34a, Cas-3, Cas-8, Cas-9 and Vimentin in A549 Cell Line. The data was presented as mean ± SD (N=3) (* = P< 0.05).
Article Snippet: Cancer Cell Cultures MCF7, MDA 231, and
Techniques: Expressing
Journal: Cancer Drug Resistance
Article Title: EGFR signaling promotes resistance to CHK1 inhibitor prexasertib in triple negative breast cancer
doi: 10.20517/cdr.2020.73
Figure Lengend Snippet: Erlotinib co-exposure with prexasertib synergistically enhanced cell killing. Viability of 3D cultures of MDA-231 (A) and MDA-468 (B) after exposure to prexasertib, erlotinib, and combinations of erlotinib and prexasertib; the combination index (CI) showed synergistic interaction (CI < 1) of erlotinib and prexasertib in both MDA-231 and MDA-468 (C)
Article Snippet: Forty athymic nude mice (
Techniques:
Journal: Cancer Drug Resistance
Article Title: EGFR signaling promotes resistance to CHK1 inhibitor prexasertib in triple negative breast cancer
doi: 10.20517/cdr.2020.73
Figure Lengend Snippet: Erlotinib co-exposure with prexasertib reduced BAD phosphorylation. MDA-231 cells treated with vehicle, 20 nmol/L prexasertib, 10 µmol/L erlotinib, or both 20 nmol/L prexasertib and 10 µmol/L erlotinib at 48 and 72 h (A); MDA-468 cells treated with vehicle, 20 nmol/L prexasertib, 10 µmol/L erlotinib, or both 20 nmol/L prexasertib and 10 µmol/L erlotinib at 48 and 72 h (B)
Article Snippet: Forty athymic nude mice (
Techniques:
Journal: Cancer Drug Resistance
Article Title: EGFR signaling promotes resistance to CHK1 inhibitor prexasertib in triple negative breast cancer
doi: 10.20517/cdr.2020.73
Figure Lengend Snippet: EGF stimulation in MDA-231 increased BAD phosphorylation. MDA-231 cells treated with vehicle, 20 nmol/L prexasertib, 50 nmol/L EGF, or both 50 nmol/L EGF and 20 nmol/L prexasertib were examined after 48 h of exposure. AKT: protein kinase B; EGF: epidermal growth factor; EGFR: epidermal growth factor receptor
Article Snippet: Forty athymic nude mice (
Techniques:
Journal: Cancer Drug Resistance
Article Title: EGFR signaling promotes resistance to CHK1 inhibitor prexasertib in triple negative breast cancer
doi: 10.20517/cdr.2020.73
Figure Lengend Snippet: Co-exposure of erlotinib and prexasertib significantly reduced tumor progression in MDA-231 and MDA-468 xenografts. MDA-231 and MDA-468 xenograft containing mice were treated with vehicle, 10 mg/kg BW prexasertib, 50 mg/kg BW erlotinib, or both prexasertib and erlotinib (A); tumor weights at sacrifice for MDA-231 and MDA-468 (B)
Article Snippet: Forty athymic nude mice (
Techniques: