mapt− Search Results


97
Quanterix p tau
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
P Tau, supplied by Quanterix, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 97 stars, based on 1 article reviews
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93
Cell Signaling Technology Inc rabbit anti ptau 214
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Rabbit Anti Ptau 214, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
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94
Cell Signaling Technology Inc anti ptau231 catalog 71429s antibodies
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Anti Ptau231 Catalog 71429s Antibodies, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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96
Cell Signaling Technology Inc rabbit anti p tau 181
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Rabbit Anti P Tau 181, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 96 stars, based on 1 article reviews
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94
Cell Signaling Technology Inc rabbit anti phospho tau
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Rabbit Anti Phospho Tau, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
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94
Cell Signaling Technology Inc cell signaling technology 20473s
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Cell Signaling Technology 20473s, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/cell signaling technology 20473s/product/Cell Signaling Technology Inc
Average 94 stars, based on 1 article reviews
cell signaling technology 20473s - by Bioz Stars, 2026-04
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95
Cell Signaling Technology Inc mouse anti tau46 antibody
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Mouse Anti Tau46 Antibody, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse anti tau46 antibody/product/Cell Signaling Technology Inc
Average 95 stars, based on 1 article reviews
mouse anti tau46 antibody - by Bioz Stars, 2026-04
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96
Cell Signaling Technology Inc ∙ rabbit anti phospho tau thr181
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
∙ Rabbit Anti Phospho Tau Thr181, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 96 stars, based on 1 article reviews
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98
AvesLabs anti tau
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Anti Tau, supplied by AvesLabs, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 98 stars, based on 1 article reviews
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93
Addgene inc full length human t40 tau in pet29b
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Full Length Human T40 Tau In Pet29b, supplied by Addgene inc, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/full length human t40 tau in pet29b/product/Addgene inc
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96
Santa Cruz Biotechnology tau mouse monoclonal santa cruz sc
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Tau Mouse Monoclonal Santa Cruz Sc, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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88
Neuromics chicken antirat tau mapt primary antibody
Boxplots <t>of</t> <t>the</t> <t>p-tau-217</t> levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.
Chicken Antirat Tau Mapt Primary Antibody, supplied by Neuromics, used in various techniques. Bioz Stars score: 88/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Boxplots of the p-tau-217 levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.

Journal: Brain Communications

Article Title: Brain-injury and Alzheimer’s disease biomarkers are elevated in patients with suspected infection and physiological derangement: importance for context-specific interpretation of Alzheimer’s biomarkers

doi: 10.1093/braincomms/fcag063

Figure Lengend Snippet: Boxplots of the p-tau-217 levels in patients with suspected sepsis (n = 24) taken on recruitment, age- and sex- matched non-infected controls (n = 20) and Alzheimer’s disease patients (n = 9). Boxplots showing the univariate distribution of plasma p-tau-217 (pg/mL) levels in participants with suspected sepsis (n = 24), age- and sex-matched non-infected controls (n = 20), and patients with Alzheimer’s disease (n = 9). Each box represents the IQR with the median shown as a horizontal line. Individual participant data points, each representing a single participant’s biomarker measurement, are overlaid as black dots. The red dashed line indicates the reported binary threshold for Aβ-positive positron emission tomography scan in Alzheimer’s disease (0.42 pg/ml). A Kruskal–Wallis test showed significant differences across groups (χ 2 = 20.886, df = 2, P < 0.001). Post-hoc pairwise comparisons using Dunn’s test with Benjamini–Hochberg adjustment showed: Alzheimer’s disease versus non-infected controls, Z = 3.957, P < 0.001; suspected sepsis versus non-infected controls, Z = −3.713, P < 0.001; Alzheimer’s disease versus suspected sepsis, Z = 1.187, P = 0.118.

Article Snippet: In the suspected sepsis samples, non-infected age- and sex-matched controls and additional ‘positive’ controls taken from Alzheimer’s patients (n = 9), p-tau-217 was quantified using the Simoa® ALZpath p-tau-217 Advantage PLUS Assay Performance run on the Quanterix HD-X Analyser.

Techniques: Infection, Clinical Proteomics, Biomarker Discovery, Positron Emission Tomography

Stacked bar graphs of the numbers of patients in each group with p-tau-217 levels in different reference ranges, demonstrating that nearly half of the patients with suspected sepsis had elevated plasma p-tau-217. Distribution of plasma p-tau-217 concentrations (pg/ml) categorized as low (<0.4 pg/ml), intermediate (0.4–0.63 pg/ml) and high (>0.63 pg/ml) across participant groups: non-infected controls (n = 20), patients with suspected sepsis (n = 24) and Alzheimer’s disease controls (n = 9). The stacked bars show the proportion of participants in each category, with percentages annotated within each segment.

Journal: Brain Communications

Article Title: Brain-injury and Alzheimer’s disease biomarkers are elevated in patients with suspected infection and physiological derangement: importance for context-specific interpretation of Alzheimer’s biomarkers

doi: 10.1093/braincomms/fcag063

Figure Lengend Snippet: Stacked bar graphs of the numbers of patients in each group with p-tau-217 levels in different reference ranges, demonstrating that nearly half of the patients with suspected sepsis had elevated plasma p-tau-217. Distribution of plasma p-tau-217 concentrations (pg/ml) categorized as low (<0.4 pg/ml), intermediate (0.4–0.63 pg/ml) and high (>0.63 pg/ml) across participant groups: non-infected controls (n = 20), patients with suspected sepsis (n = 24) and Alzheimer’s disease controls (n = 9). The stacked bars show the proportion of participants in each category, with percentages annotated within each segment.

Article Snippet: In the suspected sepsis samples, non-infected age- and sex-matched controls and additional ‘positive’ controls taken from Alzheimer’s patients (n = 9), p-tau-217 was quantified using the Simoa® ALZpath p-tau-217 Advantage PLUS Assay Performance run on the Quanterix HD-X Analyser.

Techniques: Clinical Proteomics, Infection