deferoxamine dfo Search Results


deferoxamine (dfo  (AbMole Bioscience)
90
AbMole Bioscience deferoxamine (dfo
Deferoxamine (Dfo, supplied by AbMole Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pmc10749887-49-5-10?v=AbMole+Bioscience
Average 90 stars, based on 1 article reviews
deferoxamine (dfo - by Bioz Stars, 2026-08
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deferoxamine (dfo)  (ApexBio)
90
ApexBio deferoxamine (dfo)
Deferoxamine (Dfo), supplied by ApexBio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pm39706856-223-17-20?v=ApexBio
Average 90 stars, based on 1 article reviews
deferoxamine (dfo) - by Bioz Stars, 2026-08
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deferoxamine dfo  (Cayman Chemical)
90
Cayman Chemical deferoxamine dfo
Effects of HMGB1 on rat H9c2 cardiomyoblasts. ( A – D ) Effect of HMGB1 on cell death (trypan blue staining) ( A ), apoptosis ( B ), mtHR ( C ), and MMP ( D ). ( E ) Inhibition of cell death ( F ) The effect of chloroquine on HMGB1-induced apoptosis and autophagy. ( G , H ) Effects of HMGB1 on the levels of autophagy-associated proteins ( G ) and autophagy signal-associated proteins ( H ). Right panels, semi-quantified graphs. Statistical significance was calculated using an ordinary ANOVA. Asterisk, p value. C, control; HMGB1, high-mobility group box-1 (40 μg/mL); mtHR, mitochondrial hydroxyl radical; MMP, mitochondrial membrane potential; ZVAD, Z-VAD-FMK; NAC, N-acetyl-L-cysteine; DFO, <t>deferoxamine;</t> FRS, ferrostatin-1; 4PBA, 4-phenylbutyric acid; ATG5, autophagy-Related Gene 5; LC3, microtubule-associated protein 1A/1B-light chain 3; PI3K, phosphoinositide 3-kinase; pAKT, phosphorylated AKT; CLQ, chloroquine.
Deferoxamine Dfo, supplied by Cayman Chemical, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pmc11084938-180-29-33?v=Cayman+Chemical
Average 90 stars, based on 1 article reviews
deferoxamine dfo - by Bioz Stars, 2026-08
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deferoxamine dfo m5129  (AbMole Bioscience)
90
AbMole Bioscience deferoxamine dfo m5129
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo M5129, supplied by AbMole Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pmc09867595-54-6-13?v=AbMole+Bioscience
Average 90 stars, based on 1 article reviews
deferoxamine dfo m5129 - by Bioz Stars, 2026-08
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deferoxamine (dfo) mesylate  (FUJIFILM)
90
FUJIFILM deferoxamine (dfo) mesylate
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine (Dfo) Mesylate, supplied by FUJIFILM, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pm38413363-26-24-33?v=FUJIFILM
Average 90 stars, based on 1 article reviews
deferoxamine (dfo) mesylate - by Bioz Stars, 2026-08
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dfo-toc (deferoxamine-c6-dphe-cys-tyr-dtrp-lys-thr-cys-thr-ol, peptide purity = 92.9%, molecular weight = 1901.3 g/mol)  (CPC Scientific)
90
CPC Scientific dfo-toc (deferoxamine-c6-dphe-cys-tyr-dtrp-lys-thr-cys-thr-ol, peptide purity = 92.9%, molecular weight = 1901.3 g/mol)
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Dfo Toc (Deferoxamine C6 Dphe Cys Tyr Dtrp Lys Thr Cys Thr Ol, Peptide Purity = 92.9%, Molecular Weight = 1901.3 G/Mol), supplied by CPC Scientific, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pm39693037-52-0-14?v=CPC+Scientific
Average 90 stars, based on 1 article reviews
dfo-toc (deferoxamine-c6-dphe-cys-tyr-dtrp-lys-thr-cys-thr-ol, peptide purity = 92.9%, molecular weight = 1901.3 g/mol) - by Bioz Stars, 2026-08
90/100 stars
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deferoxamine dfo t1637  (Topscience Co Ltd)
90
Topscience Co Ltd deferoxamine dfo t1637
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo T1637, supplied by Topscience Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pmc08811259-72-11-17?v=Topscience+Co+Ltd
Average 90 stars, based on 1 article reviews
deferoxamine dfo t1637 - by Bioz Stars, 2026-08
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deferoxamine dfo 150  (Microm International GmbH)
90
Microm International GmbH deferoxamine dfo 150
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo 150, supplied by Microm International GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/10__1002_slash_jbmr__5650181305-721-0-7?v=Microm+International+GmbH
Average 90 stars, based on 1 article reviews
deferoxamine dfo 150 - by Bioz Stars, 2026-08
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deferoxamine (dfo) bfc (p-ncs-bz-dfo)  (CheMatech Inc)
90
CheMatech Inc deferoxamine (dfo) bfc (p-ncs-bz-dfo)
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine (Dfo) Bfc (P Ncs Bz Dfo), supplied by CheMatech Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pmc08446777-42-1-8?v=CheMatech+Inc
Average 90 stars, based on 1 article reviews
deferoxamine (dfo) bfc (p-ncs-bz-dfo) - by Bioz Stars, 2026-08
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dfo reference standard usp deferoxamine mesylate reference standard lot h  (Pharmacopeia Inc)
90
Pharmacopeia Inc dfo reference standard usp deferoxamine mesylate reference standard lot h
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Dfo Reference Standard Usp Deferoxamine Mesylate Reference Standard Lot H, supplied by Pharmacopeia Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/10__1128_slash_aac__39__9__2023-27-1-13?v=Pharmacopeia+Inc
Average 90 stars, based on 1 article reviews
dfo reference standard usp deferoxamine mesylate reference standard lot h - by Bioz Stars, 2026-08
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deferoxamine mesylate (dfo)  (AbMole Bioscience)
90
AbMole Bioscience deferoxamine mesylate (dfo)
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Mesylate (Dfo), supplied by AbMole Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pm30141807-51-0-5?v=AbMole+Bioscience
Average 90 stars, based on 1 article reviews
deferoxamine mesylate (dfo) - by Bioz Stars, 2026-08
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deferoxamine dfo  (Pharma Science Nutrients Inc)
90
Pharma Science Nutrients Inc deferoxamine dfo
Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), <t>deferoxamine</t> (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.
Deferoxamine Dfo, supplied by Pharma Science Nutrients Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/deferoxamine+dfo/pm28935635-51-19-23?v=Pharma+Science+Nutrients+Inc
Average 90 stars, based on 1 article reviews
deferoxamine dfo - by Bioz Stars, 2026-08
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Image Search Results


Effects of HMGB1 on rat H9c2 cardiomyoblasts. ( A – D ) Effect of HMGB1 on cell death (trypan blue staining) ( A ), apoptosis ( B ), mtHR ( C ), and MMP ( D ). ( E ) Inhibition of cell death ( F ) The effect of chloroquine on HMGB1-induced apoptosis and autophagy. ( G , H ) Effects of HMGB1 on the levels of autophagy-associated proteins ( G ) and autophagy signal-associated proteins ( H ). Right panels, semi-quantified graphs. Statistical significance was calculated using an ordinary ANOVA. Asterisk, p value. C, control; HMGB1, high-mobility group box-1 (40 μg/mL); mtHR, mitochondrial hydroxyl radical; MMP, mitochondrial membrane potential; ZVAD, Z-VAD-FMK; NAC, N-acetyl-L-cysteine; DFO, deferoxamine; FRS, ferrostatin-1; 4PBA, 4-phenylbutyric acid; ATG5, autophagy-Related Gene 5; LC3, microtubule-associated protein 1A/1B-light chain 3; PI3K, phosphoinositide 3-kinase; pAKT, phosphorylated AKT; CLQ, chloroquine.

Journal: International Journal of Molecular Sciences

Article Title: Berberine Improves Cancer-Derived Myocardial Impairment in Experimental Cachexia Models by Targeting High-Mobility Group Box-1

doi: 10.3390/ijms25094735

Figure Lengend Snippet: Effects of HMGB1 on rat H9c2 cardiomyoblasts. ( A – D ) Effect of HMGB1 on cell death (trypan blue staining) ( A ), apoptosis ( B ), mtHR ( C ), and MMP ( D ). ( E ) Inhibition of cell death ( F ) The effect of chloroquine on HMGB1-induced apoptosis and autophagy. ( G , H ) Effects of HMGB1 on the levels of autophagy-associated proteins ( G ) and autophagy signal-associated proteins ( H ). Right panels, semi-quantified graphs. Statistical significance was calculated using an ordinary ANOVA. Asterisk, p value. C, control; HMGB1, high-mobility group box-1 (40 μg/mL); mtHR, mitochondrial hydroxyl radical; MMP, mitochondrial membrane potential; ZVAD, Z-VAD-FMK; NAC, N-acetyl-L-cysteine; DFO, deferoxamine; FRS, ferrostatin-1; 4PBA, 4-phenylbutyric acid; ATG5, autophagy-Related Gene 5; LC3, microtubule-associated protein 1A/1B-light chain 3; PI3K, phosphoinositide 3-kinase; pAKT, phosphorylated AKT; CLQ, chloroquine.

Article Snippet: The following inhibitors were utilized: N-acetyl-L-cysteine (NAC, 1 mM, Sigma, St. Louis, MO, USA), Z-VAD-FMK (ZVAD, 20 μM, Santa Cruz Biotechnology, Santa Cruz, CA, USA), ferrostatin-1 (FRS, 2 μM), deferoxamine (DFO, 200 μM, Cayman Chemicals, Ann Arbor, MI, USA), and 4-phenylbutyric acid (4PBA, 200 μM, WAKO).

Techniques: Staining, Inhibition, Control, Membrane

Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), deferoxamine (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.

Journal: Oxidative Medicine and Cellular Longevity

Article Title: Curcumin Induces Ferroptosis in Follicular Thyroid Cancer by Upregulating HO-1 Expression

doi: 10.1155/2023/6896790

Figure Lengend Snippet: Curcumin inhibits FTC tumorigenesis. (a) Nthy-ori-3-1 and FTC cells were treated with the indicated concentrations of curcumin for 24 h, and curcumin inhibited cell viability. Cell viability was measured using CCK-8 assays. (b–d) Curcumin suppressed the growth of FTC cells. FTC cells were treated with 0, 10, or 20 μ M curcumin and subjected to colony formation, cell migration, and invasion assays. Scale bars: 100 μ m. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the 0 μ M curcumin group. (e, f) FTC cells were either preincubated with various inhibitors, namely, ferrostatin-1 (Fer-1, 10 μ M), deferoxamine (DFO, 10 μ M), chloroquine (CQ, 5 μ M), autophagy inhibitor (3MA, 1 mM), and Z-VAD-FMK (5 μ M), for 2 h and then treated with 10 or 20 μ M curcumin (Cur) for 24 h or treated with curcumin alone. Cell viability was assessed using CCK-8 assays. ∗ p < 0.05 and ∗∗ p < 0.01 compared with the group receiving curcumin treatment alone. The data are presented as the mean ± standard error of the mean (SEM), n = 3. One-way ANOVA (b–d) and t test (e, f) were used to determine statistical significance.

Article Snippet: Erastin (M2679), ferrostatin-1 (Fer-1, M2698), and deferoxamine (DFO, M5129) were all purchased from AbMole Bioscience Inc. (Texas, USA).

Techniques: CCK-8 Assay, Migration