antinfκb Search Results


90
Huabio Inc anti-nf-κb
Anti Nf κb, supplied by Huabio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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MBL International anti-nf-kb
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Active Motif antibody: anti-nfκb p50 antibody
Antibody: Anti Nfκb P50 Antibody, supplied by Active Motif, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Active Motif anti-nfκb 40916
Anti Nfκb 40916, supplied by Active Motif, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Euromedex rabbit polyclonal anti-nfκb p65 antibody ab1604
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ELK Biotechnology anti-nfκb(p105)(ser932
Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of <t>NFκB(p105)</t> in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.
Anti Nfκb(P105)(Ser932, supplied by ELK Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Bioworld Antibodies antibodies against p-nfκb p65 p-iκbα
Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of <t>NFκB(p105)</t> in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.
Antibodies Against P Nfκb P65 P Iκbα, supplied by Bioworld Antibodies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Merck & Co hrp-labeled anti-nfκb
Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of <t>NFκB(p105)</t> in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.
Hrp Labeled Anti Nfκb, supplied by Merck & Co, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Becton Dickinson anti-nfκb (purified mouse anti- nf-κb colne20/p65
Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of <t>NFκB(p105)</t> in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.
Anti Nfκb (Purified Mouse Anti Nf κb Colne20/P65, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Merck & Co nfκb p52
Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of <t>NFκB(p105)</t> in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.
Nfκb P52, supplied by Merck & Co, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ImmunoReagents inc rabbit anti-nfκb-p65 (acetyl-k-310)
Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of <t>NFκB(p105)</t> in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.
Rabbit Anti Nfκb P65 (Acetyl K 310), supplied by ImmunoReagents inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
GeneTex anti-nfκb
Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of <t>NFκB(p105)</t> in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.
Anti Nfκb, supplied by GeneTex, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of NFκB(p105) in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.

Journal: International Journal of Molecular Sciences

Article Title: Cardioprotective Effects of the GRK2 Inhibitor Paroxetine on Isoproterenol-Induced Cardiac Remodeling by Modulating NF-κB Mediated Prohypertrophic and Profibrotic Gene Expression

doi: 10.3390/ijms242417270

Figure Lengend Snippet: Effect of paroxetine on ISO-induced NF-κB and inflammatory biomarkers. ( A ) Level of NFκB(p105) in myocardium tissues increased with ISO treatment (CH group) and was prevented by paroxetine pre-treatment (CH + P group) ( n = 6 per group). Serum levels of inflammatory biomarkers IL6 ( n = 4) ( B ) and CRP ( n = 6) ( C ) increased with ISO treatment (CH group) and were prevented by paroxetine and fluoxetine pre-treatment (CH + P and CH + F groups). All data are expressed as mean ± SEM. Differences between groups were detected using one-way ANOVA followed by Dunnett’s post hoc test. Statistically significant differences between groups are shown as * p < 0.05, *** p < 0.001. CH: cardiac hypertrophy untreated; CH + P: cardiac hypertrophy pretreated with paroxetine; CH + F: cardiac hypertrophy pretreated with fluoxetine; NFκB: Nuclear factor-kappa-light-chain-enhancer of activated B-cells; IL6: interleukin 6; CRP: C-reactive protein.

Article Snippet: Paraffin-embedded heart tissue sections (5 µm) were deparaffinized and then incubated with the following primary antibodies: anti-GRK2 (1:50) (Santa Cruz Biotechnology, Santa Cruz, CA, USA), anti-IκBα(pSer32/S36) (1:100), and anti-NFκB(p105)(Ser932) (1:100) (ELK Biotechnology Co., Wuhan, China) diluted in blocking buffer [(PBS solution and 0.3% TritonX100) and 10% fetal bovine serum] at 4 °C overnight.

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